Nat Genet 2012,44(4):413–419

Nat Genet 2012,44(4):413–419. Selleck GSK1210151A S411PubMedCentralPubMedCrossRef 70. Spaeth KE, Chen YS, Valdivia RH: The Chlamydia type III secretion system C-ring engages a chaperone-effector protein complex. PLoS Pathog 2009,5(9):e1000579.PubMedCentralPubMedCrossRef 71. Ponting CP: Chlamydial homologues of the MACPF (MAC/perforin) domain. Curr Biol 1999,9(24):R911-R913.PubMedCrossRef 72. Taylor LD, Nelson DE, Dorward DW, Whitmire WM, Caldwell HD: Biological characterization of Chlamydia trachomatis

plasticity zone MACPF domain family protein CT153. Infect Immun 2010,78(6):2691–2699.PubMedCentralPubMedCrossRef 73. Pettersson J, Nordfelth R, Dubinina E, Bergman T, Gustafsson M, ACP-196 nmr Magnusson KE, Wolf-Watz H: Modulation of virulence factor expression by pathogen target cell contact. Science 1996,273(5279):1231–1233.PubMedCrossRef 74. Parsot C, Ageron E, Penno C, Mavris M, Jamoussi K, d’Hauteville H, Sansonetti P, Demers B: A secreted anti-activator, OspD1, and its chaperone, Spa15, are involved in the control of transcription by the type III secretion apparatus activity in Shigella flexneri . Mol Microbiol 2005,56(6):1627–1635.PubMedCrossRef 75. Botteaux A, Sory MP, Biskri L, Parsot C, Allaoui A: MxiC is secreted by and controls the substrate specificity of the Shigella flexneri type III secretion apparatus. Mol Microbiol 2009,71(2):449–460.PubMedCrossRef 76. Feldman MF, Cornelis GR: The multitalented type III chaperones:

all you can do with 15 kDa. FEMS Microbiol Dabrafenib order Lett 2003,219(2):151–158.PubMedCrossRef Sucrase 77. Parsot C, Hamiaux C, Page AL: The various and varying roles of specific chaperones in type III secretion systems. Curr Opin Microbiol 2003,6(1):7–14.PubMedCrossRef 78. Agaisse H, Derre I: A C. trachomatis cloning vector and the generation of C. trachomatis strains expressing fluorescent proteins under the control of a C. trachomatis promoter. PLoS ONE 2013,8(2):e57090.PubMedCentralPubMedCrossRef Competing interests The authors declare that they have no competing interests. Authors’ contributions MdC, CM, FA, SVP, RM, and VB performed research and analyzed data. MdC, CM, FA, SVP,

and RM performed T3S assays and VB carried out the RT-qPCR assays. MdC also performed the translocation assays and helped to write the paper. JPG and MJB designed research and analyzed data. LJM designed research, analyzed data and wrote the paper. All authors read and approved the final manuscript.”
“Background Laribacter hongkongensis is a Gram-negative, facultative anaerobic, motile, S-shaped, asaccharolytic, urease-positive bacillus that belongs to the Neisseriaceae family of β-proteobacteria [1]. It was first isolated from the blood and thoracic empyema of an alcoholic liver cirrhosis patient in Hong Kong [1]. Recently, it was also recovered from the blood culture of a Korean patient with liver cirrhosis as a result of Wilson’s disease [2]. These cases make chronic liver disease a distinct possible risk factor for invasive L.

Comments are closed.